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Colima Sea Salt Review: How Much Sodium Do You Really Need?

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Sodium is essential for fluid balance, nerve signals, and a steady heartbeat, and most healthy people should not go on a low-salt diet unless their doctor tells them to. Large observational studies link the lowest heart risk with a moderate intake of about 3,000 to 5,000 mg of sodium a day, and higher risk at both very low and very high intakes. (1, 2)

If you have high blood pressure, kidney disease, or heart failure, follow your doctor’s salt advice. Either way, choose a clean, unrefined salt like Colima Sea Salt, hand-harvested and free of anti-caking agents, and cut back on the packaged and restaurant foods where most sodium hides. (3)

For almost 50 years, Americans have heard the same message: eat less salt for your heart.

For most healthy families, we believe that advice has gone too far. A low-salt diet is not the safe default it is made out to be, and the research on very low intake backs that up.

Sodium is an essential mineral, and God designed your body to need it every day. Your needs change with activity, sweat loss, climate, health conditions, medications, and the rest of your diet.

Too much sodium is a problem too, because it can raise blood pressure. Most of that excess comes from packaged and restaurant food, not from the salt you add at home.

The real question is not whether sodium is good or bad. It is how much you need, where it is coming from, and what kind of salt you choose when you add it to real food.

Is Less Salt Actually Good For You?

The first thing we need to clear up is the difference between sodium and salt.

Sodium is a mineral. Table salt is sodium chloride and is about 40% sodium by weight. One teaspoon of table salt contains roughly 2,400 mg of sodium. So when a health guideline says “2,300 mg per day,” it means 2,300 mg of sodium, not 2,300 mg of salt. (4)

According to the CDC, Americans currently consume more than 3,300 mg of sodium per day on average. Most of that does not come from the salt shaker. It comes from packaged, prepared, and restaurant foods. (3)

The current Dietary Guidelines for Americans, 2025-2030 recommend less than 2,300 mg of sodium per day for the general population age 14 and older. Importantly, the new guidelines also say that sodium and electrolytes are essential for hydration and that highly active people may benefit from additional sodium to replace what they lose in sweat. (5)

The American Heart Association recommends no more than 2,300 mg per day and describes 1,500 mg as an optimal goal for most adults, especially in the context of blood-pressure control. It also emphasizes that reducing intake by about 1,000 mg per day can improve blood pressure for many people. (6)

So is less sodium always better?

No. Sodium is essential, and individual needs matter.

Cutting back makes sense when intake is truly high, and the clearest benefit is lower blood pressure. In its 2019 report, the National Academies found high-strength evidence for chronic-disease risk reduction when sodium intake is reduced within the approximate range of 2,300 to 4,100 mg per day. (7)

Below 2,300 mg, the panel found the evidence much less certain. That is why we do not tell healthy families to chase the lowest number they can reach.

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The Truth Behind Low-Sodium Diet Science

The low-sodium debate did not come out of nowhere.

Public-health efforts to reduce sodium accelerated in the 1970s as hypertension became a major prevention target. Early recommendations leaned heavily on the relationship between sodium and blood pressure because long-term randomized trials measuring heart attacks, strokes, and death were difficult and expensive to conduct.

That limitation helped fuel legitimate debate for decades.

Some older trials showed that substantial sodium restriction lowered blood pressure more dramatically in people with hypertension than in people with normal blood pressure. A 2020 Cochrane review also found that major sodium reductions increased renin, aldosterone, adrenaline, and noradrenaline and produced small short-term increases in cholesterol and triglycerides. (8)

Those findings are worth knowing. The body responds to sodium restriction through multiple hormonal systems, and a low-sodium diet is not physiologically neutral.

But the same Cochrane review also confirmed a meaningful blood-pressure reduction in people with hypertension. And newer evidence has filled in important gaps that were still open when many of the classic “war on salt” articles were written.

So a low-salt diet is a real intervention with real effects on the body, not a harmless default. Here is where we land:

Excess sodium from processed food is worth cutting, but most healthy people should not go on a low-salt diet unless their doctor tells them to.

That is a much more useful conversation for real families.

What the Latest Science on Salt Shows

Several large observational studies have reported a J-shaped or U-shaped relationship between sodium and cardiovascular outcomes. In those studies, people consuming moderate amounts of sodium often appeared to fare better than people at either very high or very low intakes.

The best known is the PURE study, which in 2014 followed 101,945 adults in 17 countries. People with an estimated intake of 3,000 to 6,000 mg of sodium a day had a lower risk of death and heart events than those who ate more or less. (9)

Research since then points the same way. A 2025 study of 270,991 adults in China found that the lowest fifth of sodium intake carried a 17% higher risk of cardiovascular death than the middle, with the lowest risk at about 4,600 to 5,400 mg a day. (2)

A 2023 Dutch study of 1,603 adults aged 60 to 75 measured sodium with 24-hour urine collections, the method researchers trust most. Those in the lower half of intake, under about 3,600 mg a day, had roughly twice the risk of death of the middle group, and the risk was highest when low salt came with low protein. (10)

These studies are observational, and they share an honest limitation: measuring a person’s usual sodium intake is surprisingly difficult.

Many of these studies, PURE and the 2025 China study included, estimated 24-hour sodium intake from a single spot urine sample. A 2025 analysis involving more than 435,000 UK Biobank participants showed that common spot-urine formulas can create misleading associations between estimated sodium intake and cardiovascular outcomes. Earlier work comparing those estimates with repeated measured 24-hour urine collections found the same problem. (11)

When sodium is measured more carefully, the high end clearly matters.

A pooled analysis published in the New England Journal of Medicine included 10,709 generally healthy adults whose sodium intake was assessed with at least two 24-hour urine collections. During a median follow-up of 8.8 years, higher sodium excretion was associated with higher cardiovascular risk in a dose-response pattern. Each additional 1,000 mg of sodium excretion per day was associated with an 18% higher cardiovascular risk, while higher potassium excretion was associated with lower risk. (12)

Few people in those cohorts ate very little salt, though: only one in ten excreted less than about 2,100 mg a day. So this study speaks clearly to the harm of too much sodium, and it says little about whether going very low helps a healthy person.

Randomized evidence also matters.

The Salt Substitute and Stroke Study included 20,995 older adults in rural China, most of whom had a history of stroke or uncontrolled high blood pressure. Villages were assigned either regular salt or a salt substitute containing 75% sodium chloride and 25% potassium chloride. Over nearly five years, the salt-substitute group had lower rates of stroke, major cardiovascular events, and death. (13)

Because that intervention both lowered sodium and increased potassium, we cannot say sodium reduction alone caused every benefit. But it is powerful evidence that replacing some sodium chloride with potassium chloride can improve major cardiovascular outcomes in an appropriate high-risk population.

A 2024 systematic review and meta-analysis of longer-term randomized salt-substitution trials likewise found that salt substitutes may reduce cardiovascular and all-cause mortality, although the authors noted that evidence for some outcomes and generalizability to Western populations remained uncertain. (14)

So is eating less salt more harmful to your heart?

Very low salt intake has not been shown to protect a healthy person’s heart, and large studies link it with higher risk.

These studies show a link, not proof, and people who are already sick sometimes eat less, which can skew the results. Still, sodium guidelines were set without high-quality trials showing that low intake prevents heart events compared with average intake. (1)

That is why we tell healthy families not to go low-salt unless their doctor says so. Moderate salt on real food is the sensible middle.

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How Much Salt Should You Eat?

So should you eat more salt or less?

For most people, the best place to begin is not by chasing an extreme number. Start by looking at your actual diet.

The current federal recommendation for people age 14 and older is less than 2,300 mg of sodium per day. That is roughly the amount of sodium in one teaspoon of table salt, but remember that your daily total includes sodium already present in food. (5, 4)

The National Academies lists 1,500 mg per day as the Adequate Intake for most adults and recommends reducing intake when it is above 2,300 mg per day to lower chronic-disease risk. That does not mean 1,500 mg is a mandatory therapeutic target for every healthy adult.

We have called 3,000 to 6,000 mg per day the “Goldilocks” range, and large observational studies still point to a moderate middle, roughly 3,000 to 5,000 mg, as the lowest-risk zone. (1)

The best-measured studies are a fair reminder that more is not better. So treat the upper end as a ceiling, not a goal. (12)

But there are important exceptions to one-size-fits-all restriction.

When You May Need More Sodium

People who exercise hard, work outdoors in hot weather, sweat heavily, or lose substantial fluids can have higher sodium needs. The 2025-2030 Dietary Guidelines specifically acknowledge that highly active individuals may benefit from increased sodium intake to offset sweat losses. (5)

Prolonged endurance exercise can also create a very different electrolyte problem from the everyday high-sodium diet. Exercise-associated hyponatremia can occur when blood sodium becomes too diluted, often because fluid intake exceeds the body’s ability to excrete water, sometimes combined with sodium losses in sweat. (15)

This is why “low blood sodium” and “eating a low-sodium diet” are not interchangeable ideas.

Hyponatremia is a medical condition involving the sodium concentration in your blood. It can occur because of excess water intake, certain medications, kidney or hormonal problems, prolonged exercise, illness, and other causes. Simply eating less than 2,300 mg of sodium does not automatically mean your blood sodium will become dangerously low.

If you have high blood pressure, kidney disease, or heart failure, or take a medicine that affects sodium, follow your doctor’s salt advice. Even then, the salt you do use can be a clean, unrefined one.

Bottom line: sodium is essential, but your ideal intake is not determined by an internet slogan.

Your body needs sodium for things like:

  • Maintaining normal fluid balance
  • Supporting nerve impulses
  • Helping muscles, including the heart, contract normally
  • Supporting normal extracellular fluid volume

There is a reason humans have valued salt for thousands of years. We need sodium and chloride to live.

But needing sodium does not mean unlimited sodium is beneficial. The goal is enough for your needs without letting processed-food sodium quietly dominate your diet.

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Not All Salt Is Made Equal!

This is where our family’s preference has not changed.

When we add salt to our food, we want a salt we enjoy using, from a source we trust, with as little unnecessary processing as possible.

Standard table salt is processed to create a fine, uniform crystal. Depending on the product, it may contain anti-caking agents to keep it free-flowing. It may also be iodized, which is an important nutritional advantage for households that do not get dependable iodine elsewhere.

Sea salt is made by evaporating seawater or saltwater brine and is often less processed. It can retain trace amounts of minerals such as magnesium, potassium, and calcium. Compared with heavily refined table salt, which is stripped down to sodium chloride, unrefined sea salt like Colima is relatively rich in trace minerals and macro-minerals such as magnesium, potassium, and calcium. It is still salt, not a mineral supplement, but it is the better salt. Table salt, kosher salt, and most sea salts are still approximately 40% sodium by weight. (4)

So why do we still strongly recommend Colima Sea Salt?

Because food quality is about more than one nutrient number.

Colima Sea Salt comes from the Cuyutlán Lagoon saltpans of Colima, Mexico and is hand-harvested using traditional methods. We love its coarse texture, clean salty flavor, and the fact that it is sold without the anti-caking agents found in many conventional salts.

Ava Jane’s Kitchen also reports that its Colima Sea Salt has been laboratory tested and found free of ocean-borne microplastics. That is a manufacturer-specific testing claim, not evidence that all lagoon salt is microplastic-free or that trace minerals cancel out the cardiovascular effects of excessive sodium. (16)

One more thing matters if Colima or another specialty salt is the only salt your family uses: most specialty sea salts are not iodized. NIH notes that iodized salt is a dependable iodine source, while noniodized sea salt provides virtually no iodine. Make sure your family gets adequate iodine from another reliable food or supplement source if your salt does not provide it. (17)

That is how we look at salt today.

Do not fear real salt on God-made food, and do not put your family on a low-salt diet unless your doctor tells you to. Just do not let packaged food manufacturers choose most of your sodium for you.

Cook from scratch. Taste your food. Use a salt you trust. Pay attention to your blood pressure and individual needs. That is a much wiser strategy than turning salt into either a villain or a miracle food.

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Dangers of a Low-Sodium Diet: Benefits of Colima Sea Salt

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Keep reading: 7 Heart Healthy Lifestyle Changes to Transform Your Health · Essential Oils for High Blood Pressure: Aromatherapy Inhaler Recipe · Essential Oils for Heart Health: Best Oils, Benefits & Recipes

References

  1. Mente A, O’Donnell M, Yusuf S. Sodium Intake and Health: What Should We Recommend Based on the Current Evidence? Nutrients. 2021. Source.
  2. Tian Y, Lin C, Zhong H, et al. Associations and mediators of estimated sodium intake with cardiovascular mortality: data based on a national population cohort. BMC Medicine. 2025. Source.
  3. Centers for Disease Control and Prevention. “About Sodium and Health.” Updated June 29, 2026. Source.
  4. American Heart Association. “Sodium Sources: Where Does All That Sodium Come From?” Last reviewed July 15, 2025. Source.
  5. U.S. Department of Health and Human Services and U.S. Department of Agriculture. Dietary Guidelines for Americans, 2025-2030. 10th Edition. January 2026. Current Dietary Guidelines.
  6. American Heart Association. “How Much Sodium Should I Eat Per Day?” Source.
  7. National Academies of Sciences, Engineering, and Medicine. Dietary Reference Intakes for Sodium and Potassium. Washington, DC: National Academies Press; 2019. doi:10.17226/25353.
  8. Graudal NA, Hubeck-Graudal T, Jürgens G. “Effects of low sodium diet versus high sodium diet on blood pressure, renin, aldosterone, catecholamines, cholesterol, and triglyceride.” Cochrane Database of Systematic Reviews. 2020;12:CD004022. doi:10.1002/14651858.CD004022.pub5.
  9. O’Donnell M, Mente A, Rangarajan S, et al. Urinary sodium and potassium excretion, mortality, and cardiovascular events. New England Journal of Medicine. 2014. Source.
  10. Hessels NR, Zhu Y, Bakker SJL, et al. Low Sodium Intake, Low Protein Intake, and Excess Mortality in an Older Dutch General Population Cohort: Findings in the Prospective Lifelines-MINUTHE Study. Nutrients. 2023. Source.
  11. Song J, Wang C, Pombo-Rodrigues S, MacGregor GA, Campbell NRC, He FJ. “Formulas to estimate dietary sodium intake from spot urine lead to misleading associations with cardiovascular disease risk and mortality.” Journal of Hypertension. 2025;43(4):681-689. doi:10.1097/HJH.0000000000003959.
  12. Ma Y, He FJ, Sun Q, et al. “24-Hour Urinary Sodium and Potassium Excretion and Cardiovascular Risk.” New England Journal of Medicine. 2022;386:252-263. doi:10.1056/NEJMoa2109794.
  13. Neal B, Wu Y, Feng X, et al. “Effect of Salt Substitution on Cardiovascular Events and Death.” New England Journal of Medicine. 2021;385:1067-1077. doi:10.1056/NEJMoa2105675.
  14. Greenwood H, Barnes K, Clark J, Ball L, Albarqouni L. “Long-Term Effect of Salt Substitution for Cardiovascular Outcomes: A Systematic Review and Meta-analysis.” Annals of Internal Medicine. 2024;177(5):643-655. doi:10.7326/M23-2626.
  15. Hew-Butler T. “Exercise-Associated Hyponatremia.” Frontiers of Hormone Research. 2019;52:178-189. doi:10.1159/000493247.
  16. Ava Jane’s Kitchen. “Colima Sea Salt.” Product and manufacturer testing information. Source.
  17. National Institutes of Health, Office of Dietary Supplements. “Iodine: Health Professional Fact Sheet.” Source.

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